WHAT ARE TELOMERES?
Telomeres are segments of DNA which protects the end of our chromosomes when cells divide.
Think like the plastic tips of shoelaces that keep the laces together. Telomeres function similarly, preventing chromosomes from fraying or tangling with one another. When that happens, it can cause genetic information to get mixed up, duplicated, destroyed or leading to cell malfunction. As a result, the telomeres become shorter until the cells are unable to divide further and become inactive or die. This is how we age.
The rate at which telomeres shorten is accelerated by:
- Oxidative Stress
- Nutritional Deficiencies
- Genetic Pre-Disposition
- and other lifestyle habits
As quicker and faster the length of our telomeres shortness happens, the higher the chance of cell aging, cell death, cardiovascular disease, dementia, stroke, and cancers.
HOW IS MICRONUTRIENT STATUS LINKED TO TELOMERE LENGTH?
Micronutrient status, levels of Omega-3 fatty acids and antioxidant capacity has direct implications for telomere length.
The synergistic effect of micronutrients on the various bodily systems creates a complex web of metabolic pathways that can be profoundly affected by a single nutrient deficiency. This makes it especially important to correct specific deficiencies and maintain micronutrient balance.
Recent studies suggest that high levels of: Homocysteine, Blood Sugar, HA1C, Insulin, Cholesterol, CRP as well as low Vitamin D are associated with telomere
TELOMERE LENGTH AND CANCER
Telomere length shortens with age. Progressive shortening of telomeres leads to senescence, apoptosis, or oncogenic transformation of somatic cells, affecting the health and lifespan of an individual.
Shorter telomeres have been associated with increased incidence of diseases and poor survival.
The rate of telomere shortening can be either increased or decreased by specific nutrition and lifestyle.
Better choice of diet and activities has a greatest potential to reduce the rate of telomere shortening or at least prevent excessive telomere attrition, leading to delayed onset of age-associated diseases and increased lifespan.
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